Patient, male , 55 years old who underwent a renal transplantation on August 2002
On October 2007, because of a chronic transplant nephropathy restarted haemodialysis.
Then the immunosuppressive therapy , sirolimus and cyclosporine, was discontinued and steroid was decreased.
On February 2008 was admitted in the hospital due to haematuria, pain of the graft, fatigue and anemia.
Gray scale ultrasonographic findings demonstrate renal enlargement, decreased echogenicity of the renal cortex, loss of corticomedullary differentiation, prominent pyramids, and effacement of the central sinus echo complex.
Color Doppler ultrasonography findings demonstrate scarce parenchymal vascularity and interlobar arteries with normal systolic flow and scarce or absence diastolic flow
( Increased I.R. > 0.8 ). [video1]

A bolus of Methilprednisolon was performed which improved the clinical picture.
The steroid therapy was continued at 5 mg per day per os and the patient was discharged from the hospital
On February 14 the patient was reametted at the hospital because of fatigue, fever, pain of the graft and anemia.
CRP was 15 mg/dl and WC 12.000/mm�.
At this point the patient was admitted in angiography for the embolization of the transplanted kidney. [video2]

After embolization the examination of Color Doppler ultrasound shows a total absence of vascular flow in the transplanted kidney. [video3]

Three days after the procedure, he was discharged with the resolution of the clinical picture.